Food made from fresh fruits that are sun-dried or dried. The moisture content is less than 12%, which is easy to preserve. For direct consumption or reproduction of food. A food made from peach, apricot, plum, jujube or melon, ginger and other fruits and vegetables, and then processed with sugar or honey. In addition to being eaten directly as a snack or snack, candied fruit can also be used as a decoration on cakes, biscuits and other snacks. Fruit Slices,Dried Blueberry,Dried Bayberry,Banana Slices Hangzhou Aiyomi food co.,LTD , https://www.aiyomisnacks.com In recent years, there have been a number of areas in which chickens have low feed intake, poor growth, weight loss, poor uniformity, faeces and other symptoms, glandular and gastric enlargement such as globular, glandular and gastric mucosal ulcers, shedding, and muscle and stomach erosion as the main features. In the epidemic, the mortality rate of infected chickens can reach 30% to 50%, and the disease duration and mortality rate are high, and the incidence rate is on the rise. First, the popular features: 1. Glandular gastritis can occur in different breeds of hens and broilers, with multiple broiler chickens, broiler chickens and young chickens, followed by meat cockerels and hybrid broilers. 2. The disease has a wide epidemic. Incidence in the affected areas is generally 7% to 28%, and the mortality rate is generally 30% to 50%. The earliest age of onset was seen on 5- to 7-day-old days, and the symptoms on the 7th to 15th day of age were obvious. After the 25th day of age, the incidence of chickens was significantly decreased. The laying hens were also affected. The disease duration was 10 to 15 days. Up to 35 days, 5 to 8 days after the onset of the death peak. Second, caused by glandular gastritis and muscle and stomach erosion causes: 1, non-infectious factors: (1) feed malnutrition, excessive copper sulfate, dietary amino acid imbalance, dietary biogenic amines (such as fish meal, corn screening products Prebiotics, bean cakes, vitamin premixes, fats, poultry meal, and meat and bone meal contain high levels of biogenic amines, lack of dietary fiber, fasting, and water cuts are all prone to this disease; (2) excessive stocking density, chicks Poor early brooding, long time chick transportation, dehydration, etc. are the causes of the disease. (3) Mycotoxins, etc. 2. Infectious factors: (1) Chicken pox, especially eye-type fowlpox (characterized by blinking), is an important cause of the onset of glandular gastritis; (2) Unexplained ophthalmia: such as infectious bronchitis, infectious throat Oral inflammation caused by bronchitis, various bacteria, vitamin A deficiency or poor ventilation can lead to the onset of glandular gastritis; (3) Some of the vertically transmitted unknown pathogens or Marek’s disease vaccines that have been contaminated by specific pathogens are probably the The main pathogens of the disease, such as chicken reticuloendotheliosis (REV), chicken anemia factor (CAV), etc.; (4) adenovirus inclusion bodies of epithelial cells, reovirus infection is the factor in the pathogenesis of histamine One (5) anaerobic bacteria, such as Clostridium, are sometimes secondary infections of ulcerative colitis and necrotic hepatitis. Third, the clinical symptoms of glandular gastritis and muscle and stomach erosion: (1) The chickens in the early stage of the disease are mad rushing, the feed intake is decreased, and the chicken manure is eaten on the Internet; (2) The diseased chickens are followed by drowsy and necking. Sluggishness, feed intake is only 1/2-1/3 of normal food intake, sluggish growth or stagnation of chickens leads to large unevenness of the uniformity of the group, the size varies greatly; the average body weight is only 2/3-1 of the standard body weight /2, lying in the ground; there is no chicken activity in the central part of the severe chicken net racks, and large groups are squashed on both sides of the house to be warm; (3) feathers are loose, and crowns are pale and shrinking. Visible mucous membranes, dry skin on the legs, cool to touch; (4) large swarms of feces are strips; chicken body weight loss, diarrhea, individual row brown to red to black feces, blood in the feces sometimes, (5) The morbidity is very fast. At the beginning, only individual snoring can be achieved. Within three days, it can reach more than 80% of large groups. Severe persons became unconscious until death. Pathological changes: (1) Swelling of the intestine, dark brownish water content of the contents, catarrhal and hemorrhagic inflammation in the duodenum and cecum, enlargement of the liver and heart, soft texture, and kidney disease in some dead chickens Swelling, deposition of urate; (2) Swollen or olive-like glandular stomach wall swelling, severe spherical, irregular glandular protrusion, deformation, gently pressurizing the milky liquid can be extruded, glandular stomach nipple The edema was hard-like, with serrated scissors scraping the serrated resistance, partially white gel-like, severe keratinocyte membrane ulcers, myogastric membrane hemorrhage; (3) the diameter of myogastric stomach became thicker, the length was shortened, was a cucurbit mouth or Garlic head, the appearance of a clear red and white coagulation necrosis or spot, and the junction of the gland and stomach were different degrees of erosion, ulcers, muscle wall swelling, thickening of the muscle wall incision is more obvious, muscle keratinocyte membrane Black-green or black, bark-like appearance, with erosion areas and ulcer lesions, stratum corneum is not easy to peel; (4) diseased chicken showed weight loss, pale muscle, thymus, Fabricius atrophy, hernia sac expansion, there is black Brown rice soup-like; gallbladder dilated to dark green, bile overflow; (5) clinical glandular gastritis Complicated by other symptoms such as liver and kidney syndrome, bursal damage and even coccidia infection. If you miss the opportunity for treatment, it will be secondary to E. coli and other diseases. V. Prevention: The disease is not a good treatment program, the first: mainly the virus Clostridium welchii; second: taking into account mycotoxins six, treatment experience experience: the disease was treated well before twenty days, A course of treatment is usually four days. After the third day of medication, the feed intake will increase rapidly.
Five black chicken green shell egg chicken glandular gastritis and muscle and stomach erosion
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